The smell of EBOLA

If it were possible, I would spend full time on the research of the Cytokine Cycle and it dysfunction. Unfortunately my capital is low and I will have to do the best I can to post on 3 jobs. I am not complaining, but I do need to focus. Here in part is this effort

The first point of clarity is that Ebola is fatal yet HIV is not at this time due to good treatment options including One a day treatment. the only similarity is that they induce dysfunctions of the immune system as previously reported. Prior to the control of the propagation of the HIV virus is had been said that you could smell when the virus was high and immune function low due to massive amounts of Interferon Gamma being produced and the smell of the person with the infect. The smell was due to neopterin. It is almost like the old days when a doctor needed to do a good exam first. Part of this exam will be to smell your suspected patients with fever. You will never again forget the smell of neopterin. I will speak more on this later

EBOLA FEAR

With all the posturing and questioning on in the media, there is little information posted as to how the Ebola virus kills and what steps might be taken in order to prevent mortality.

One way is to simply study the normal cytokine cycle and determine how the components that are elevated in fatal cases (Neopterin, TNF, which is is indicative of Interferon breakdown ) can be regulated back to homeostatic controls.

As has been said previously, all disease is the result of the the removal of homeostatic controls in IL1. TNF NEGATIVE FEEDBACK inhibition. For the IL1 , IL6 , TNF-alpha to remain elevated implies that something is disallowing their being turned off

Here is an article for the strong in heart to help decipher this enigmatic problem

.http://bit.ly/1su2Jj0


Here is another article attempting to emphasize the importance of study of the pathways of inflammatory cytokines in order to understand how they can become pathogenic

http://bit.ly/1yHisQM

One thing is for sure. Tumor Necrosis Factor can kill at minimally elevated levels. Whatever pathogenesis that allows it to be elevated in HIV or EBOLA can be eradicated by giving monoclonal antibody to TNF-alpha. This may stop the shock that it causes as well as the loss of endothelial layer integrity and loss of plasma protein to the third space